Dopa-decarboxylation in the striata of rats with unilateral substantia nigra lesions

Neurosci Lett. 1992 Nov 23;147(1):53-7. doi: 10.1016/0304-3940(92)90773-z.

Abstract

The source and site of the DOPA decarboxylation to dopamine in Parkinson's disease (PD) and animal models of PD are controversial. Since most of aromatic L-amino acid decarboxylase (AADC) are lost along with the degenerating dopaminergic neurons, we addressed the possibility that other decarboxylases or a novel protein that is structurally different from AADC decarboxylate L-DOPA in the denervated striatum. Immunotitration of the extracts from the denervated striatum with AADC antibody showed that all activity can be attributed to AADC-immunoreactive protein. We then investigated if there are non-dopaminergic intrinsic striatal neurons that express AADC. No evidence of such neurons was noted by immunocytochemistry and in situ hybridization.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Aromatic-L-Amino-Acid Decarboxylases / metabolism
  • Corpus Striatum / enzymology
  • Corpus Striatum / metabolism*
  • Decarboxylation
  • Denervation
  • Dialysis
  • Dihydroxyphenylalanine / metabolism*
  • Dopa Decarboxylase / metabolism
  • Immunohistochemistry
  • In Situ Hybridization
  • Male
  • Rats
  • Substantia Nigra / enzymology
  • Substantia Nigra / metabolism*

Substances

  • Dihydroxyphenylalanine
  • Dopa Decarboxylase
  • Aromatic-L-Amino-Acid Decarboxylases