Thioredoxin secreted upon ultraviolet A irradiation modulates activities of matrix metalloproteinase-2 and tissue inhibitor of metalloproteinase-2 in human dermal fibroblasts

Arch Biochem Biophys. 2004 Mar 1;423(1):218-26. doi: 10.1016/j.abb.2003.12.026.

Abstract

Regulation of the balance of matrix metalloproteinase-2 (MMP-2) and its tissue inhibitor (TIMP-2) by thioredoxin (Trx) was investigated in human dermal fibroblasts. Expression and secretion of Trx and Trx reductase 1 (TR1) was increased after ultraviolet (UV) A irradiation. A significant increase in proMMP-2 activity and a decrease of TIMP-2 activity in supernatants of UVA-irradiated fibroblasts were observed in gelatin and reverse zymography compared to non-irradiated fibroblasts. Removal of Trx or TR1 by immunoprecipitation diminished these changes in proMMP-2 activity. Incubation with 5, 5'-dithio-bis-2-nitrobenzoic acid (DTNB) also suppressed these changes. Incubation with recombinant Trx or TR decreased TIMP-2 activity and increased MMP-2 activity. UVA-irradiated fibroblasts, transiently transfected with a dominant-negative mutant or wild-type Trx, showed down- or upregulation of proMMP-2 activities, respectively, without significant change of protein amount. In conclusion, thioredoxin secreted by UVA irradiation is involved in the regulation of MMP-2 and TIMP-2 activities through its redox activity in human dermal fibroblasts.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Fibroblasts / metabolism
  • Fibroblasts / radiation effects
  • Humans
  • Matrix Metalloproteinase 2 / metabolism*
  • Skin / enzymology
  • Skin / metabolism*
  • Skin / radiation effects
  • Thioredoxins / metabolism*
  • Thioredoxins / radiation effects
  • Tissue Inhibitor of Metalloproteinase-2 / metabolism*
  • Ultraviolet Rays*

Substances

  • Tissue Inhibitor of Metalloproteinase-2
  • Thioredoxins
  • Matrix Metalloproteinase 2