Inhibitors of human renin with C-termini derived from amides and esters of alpha-mercaptoalkanoic acids

J Med Chem. 1992 Jul 24;35(15):2772-81. doi: 10.1021/jm00093a009.

Abstract

New transition-state analogues bearing C-termini derived from alpha-mercaptoalkanoic acids, esters, and amides were prepared and evaluated as inhibitors of human renin. Addition of alpha-mercaptoalkanoate esters to a chiral Boc-amino epoxide intermediate led ultimately to the target [(2R,3S)-3-(BocPheHis-amino)-4-cyclohexyl-2-hydroxy-1-butyl]thio derivatives. The corresponding sulfoxide and sulfone analogues were also investigated. Some of these derivatives, including one with a stable BocPhe replacement, were relatively potent inhibitors of human plasma renin, having IC50 values below 10 nM. When selected compounds were administered intravenously to sodium-deficient rhesus monkeys (Macaca mulatta) at 0.06-1 mg/kg, they reduced plasma renin activity by 87-94%. However, the accompanying drop in blood pressure was of short duration.

MeSH terms

  • Amides / chemistry
  • Amides / pharmacology
  • Animals
  • Blood Pressure / drug effects
  • Esters / chemistry
  • Esters / pharmacology
  • Female
  • Heart Rate / drug effects
  • Humans
  • Macaca mulatta
  • Male
  • Molecular Structure
  • Renin / antagonists & inhibitors*
  • Renin / blood
  • Sulfhydryl Compounds / chemistry
  • Sulfhydryl Compounds / pharmacology*

Substances

  • Amides
  • Esters
  • Sulfhydryl Compounds
  • Renin