Effect of pH on quercetin-induced suppression of heat shock gene expression and thermotolerance development in HT-29 cells

Biochem Biophys Res Commun. 1992 Jul 31;186(2):1121-8. doi: 10.1016/0006-291x(92)90863-g.

Abstract

When cells were heated for 15 min at 45 degrees C, they became thermotolerant to a second heat exposure at 45 degrees C. Thermotolerance developed rapidly, reached its maximum 6 hr after heat shock, and then gradually decayed. The development of thermotolerance was partially suppressed by treatment with various concentrations of quercetin (0.05-0.2 mM) at pH 7.4 after the initial heat treatment. In contrast, the drug markedly inhibited thermotolerance development at pH 6.5. Furthermore, a combination of low pH and quercetin treatment distinctively altered the expression of HSP70 gene compared with that of HSP28 or HSP90 gene. These results demonstrate a good correlation between the amount of HSP70 gene expression and development of thermotolerance.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Blotting, Northern
  • Cell Line
  • Colonic Neoplasms
  • DNA Probes
  • Dose-Response Relationship, Drug
  • Gene Expression / drug effects
  • Heat-Shock Proteins / biosynthesis
  • Heat-Shock Proteins / genetics*
  • Hot Temperature
  • Humans
  • Hydrogen-Ion Concentration
  • Kinetics
  • Quercetin / pharmacology*
  • RNA, Messenger / genetics
  • RNA, Messenger / isolation & purification
  • RNA, Messenger / metabolism
  • Temperature

Substances

  • DNA Probes
  • Heat-Shock Proteins
  • RNA, Messenger
  • Quercetin