Regulation of heme pathway in regenerating mouse liver

Comp Biochem Physiol B. 1992 Jan-Feb;101(1-2):243-6. doi: 10.1016/0305-0491(92)90186-u.

Abstract

1. delta-Aminolevulinic acid synthetase (ALA-S), rhodanese and microsomal heme oxygenase (MHO), were quantitated in Cl4C induced regenerating mouse liver. 2. Maximal hepatomegalia was observed at 48 hr after i.p. injection of a single dose of the toxin. 3. ALA-S activity decreased on day 2, and then significantly increased (50%) between days 3 and 7, returning afterwards to control values. 4. Cytoplasmic rhodanese, as well as MHO activities, exhibited a clear correlation as compared with the ALA-S activity profile. 5. Porphyrin biosynthesis from precursor delta-aminolevulinic acid (ALA) was significantly increased even after 15 days of intoxication. 6. Present results would indicate that Cl4C is acting in a dual fashion.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 5-Aminolevulinate Synthetase / metabolism
  • Aminolevulinic Acid / metabolism
  • Animals
  • Carbon Tetrachloride / pharmacology
  • Cytochrome P-450 Enzyme System / deficiency
  • Heme / metabolism*
  • Heme Oxygenase (Decyclizing) / metabolism
  • Kinetics
  • Liver / drug effects
  • Liver / enzymology
  • Liver / metabolism*
  • Liver Regeneration / drug effects
  • Liver Regeneration / physiology*
  • Mice
  • Microsomes, Liver / drug effects
  • Microsomes, Liver / enzymology
  • Porphyrins / biosynthesis
  • Thiosulfate Sulfurtransferase / metabolism

Substances

  • Porphyrins
  • Heme
  • Aminolevulinic Acid
  • Cytochrome P-450 Enzyme System
  • Carbon Tetrachloride
  • Heme Oxygenase (Decyclizing)
  • 5-Aminolevulinate Synthetase
  • Thiosulfate Sulfurtransferase