Expression pattern of vascular endothelial growth factor-C in human colorectal normal mucosa and neoplastic mucosa

Hepatogastroenterology. 2004 Mar-Apr;51(56):391-5.

Abstract

Background/aims: Vascular endothelial growth factor-C (VEGF-C) is a potent growth factor stimulating lymphangiogenesis.

Methodology: We examined the expression of VEGF-C immunohistochemically in neoplastic as well as normal mucosa of colorectal tissues, and evaluated the significance of VEGF-C in colorectal carcinogenesis and as a marker to predict the outcome of colorectal cancer.

Results: VEGF-C was strongly stained in 70/79 adenomas (89%), but the staining was focal in all cases, and the expression pattern in adenomas was not significantly related to either dysplasia or size of the adenoma. In the 8/8 intramucosal carcinomas within adenomas, both the carcinomatous and adenomatous lesions were stained focally, but in 6 cases (75%), the VEGF-C-positive area was larger in the carcinomatous lesion than in the adenomatous lesion. In most invasive adenocarcinomas, VEGF-C was clearly stained (83/85; 98%), with both a focal (40%) and diffuse (60%) staining pattern. In invasive carcinomas, the expression of VEGF-C was significantly correlated with lymphatic involvement, lymph node metastasis and tumor size, but not with venous involvement or liver metastasis. Survival rate tended to be lower in the high VEGF-C group than in the low group, although statistical significance was not observed.

Conclusions: These results suggest that VEGF-C plays a positive role in lymphatic spread in colorectal carcinomas.

MeSH terms

  • Adenocarcinoma / metabolism
  • Adenoma / metabolism
  • Aged
  • Colonic Polyps / metabolism
  • Colorectal Neoplasms / metabolism*
  • Female
  • Humans
  • Immunoenzyme Techniques
  • Immunohistochemistry
  • Intestinal Mucosa / metabolism*
  • Lymphatic Metastasis
  • Male
  • Middle Aged
  • Tumor Suppressor Protein p53 / metabolism
  • Vascular Endothelial Growth Factor C / metabolism*
  • Vascular Endothelial Growth Factor Receptor-3 / metabolism

Substances

  • Tumor Suppressor Protein p53
  • Vascular Endothelial Growth Factor C
  • Vascular Endothelial Growth Factor Receptor-3