Generation and characterization of a cell line with inducible expression of Ca(v)3.2 (T-type) channels

Assay Drug Dev Technol. 2003 Oct;1(5):637-45. doi: 10.1089/154065803770380995.

Abstract

Establishment of stable cell lines that constitutively express Ca(2+) channels at high density and that are useful for in vitro studies may be complicated by problems with seal quality and duration during whole-cell patch-clamp electrophysiology. The current studies describe the generation and characterization of cells that express the human alpha1H T-type Ca(2+) channel under the control of a tetracycline-inducible expression system. Western blot and immunostaining studies revealed that expression of the alpha1H protein occurred only in the presence of tetracycline. Using the whole-cell patch-clamp method, the cells displayed peak inward currents of 1.15 +/- 0.14 nA in response to voltage-clamp steps. The T-type Ca(2+) current was inhibited by the T-type Ca(2+) channel antagonist, mibefradil, with an IC(50) of 160 nM. This cell line, with inducible channel expression, sealed with longer duration during whole-cell patch-clamp recording when compared with a cell line that constitutively expresses the alpha1H Ca(2+) channel. Ca(2+) influx through this channel could also be detected after the addition of extracellular Ca(2+). The amount of Ca(2+) influx was dependent on the [Ca](o) with an EC(50) of 4 mM. The Ca(2+) influx was also inhibited by mibefradil with a potency (IC(50) = 183 nM) similar to that observed in the voltage-clamp studies. Overall, this inducible alpha1H Ca(2+) channel-expressing cell line is useful for the study of human T-type Ca(2+) channel function, and offers advantages over a similar cell line that constitutively expresses the channel.

MeSH terms

  • Calcium / metabolism*
  • Calcium Channel Blockers / pharmacology*
  • Calcium Channels, T-Type / drug effects
  • Calcium Channels, T-Type / physiology*
  • Cell Culture Techniques / methods
  • Cell Line
  • Cells, Cultured
  • Humans
  • Ion Channel Gating / drug effects
  • Kidney / drug effects
  • Kidney / embryology
  • Kidney / physiology*
  • Membrane Potentials / drug effects
  • Membrane Potentials / physiology*
  • Protein Engineering / methods*
  • Recombinant Proteins / biosynthesis
  • Recombinant Proteins / metabolism
  • Tetracycline / pharmacology*

Substances

  • CACNA1H protein, human
  • Calcium Channel Blockers
  • Calcium Channels, T-Type
  • Recombinant Proteins
  • Tetracycline
  • Calcium