The cystine-glutamate transporter in the accumbens: a novel role in cocaine relapse

Trends Neurosci. 2004 Feb;27(2):74-6. doi: 10.1016/j.tins.2003.11.007.

Abstract

Baker et al. have recently studied the potential role of cocaine-induced alterations in accumbens cystine-glutamate transporter activity (which controls basal extracellular glutamate levels) during cocaine-induced relapse to drug seeking in rats. Their data provide new evidence that neuroadaptations induced by repeated exposure to cocaine and subsequent withdrawal can play a causal role in drug relapse. These data also suggest the cystine-glutamate transporter as a novel target for medication that could prevent cocaine relapse.

Publication types

  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Adaptation, Physiological
  • Amino Acid Transport System y+*
  • Animals
  • Carrier Proteins / physiology*
  • Cocaine-Related Disorders / physiopathology*
  • Humans
  • Neurons / enzymology
  • Nucleus Accumbens / cytology
  • Nucleus Accumbens / enzymology*
  • Rats
  • Receptors, AMPA / metabolism
  • Receptors, N-Methyl-D-Aspartate / metabolism
  • Recurrence

Substances

  • Amino Acid Transport System y+
  • Carrier Proteins
  • Receptors, AMPA
  • Receptors, N-Methyl-D-Aspartate
  • SLC7A11 protein, human