Role of Ca2+ in induction of neurotransmitter-related gene expression by butyrate

Neuroreport. 2004 May 19;15(7):1177-81. doi: 10.1097/00001756-200405190-00019.

Abstract

We examined the effect of butyrate on neurotransmitter-related gene expression and calcium homeostasis in PC12 cells. Pretreatment with Ca2+ chelators (EGTA or BAPTA-AM) attenuated the butyrate-triggered accumulation of TH and ppEnk mRNA indicating that Ca2+ plays a role in butyrate-induced regulation of neuronal genes. Butyrate alone did not alter intracellular Ca2+ levels as determined by Fura-PE3 fluorescence; however, pretreatment with butyrate (18-24 h) reduced the first Ca2+ peak and prevented the second sustained rise in [Ca2+]i as induced by nicotine or ryanodine. In contrast, butyrate had no effect on Ca2+ transients when added shortly before or during nicotine or ryanodine stimulation. These results suggest that chronic butyrate exposure can modulate cell responses by affecting intracellular Ca2+ signaling.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Butyrates / pharmacology*
  • Calcium / physiology*
  • Gene Expression Regulation / drug effects*
  • Gene Expression Regulation / physiology
  • Intracellular Fluid / drug effects
  • Intracellular Fluid / metabolism
  • Intracellular Fluid / physiology
  • Neurotransmitter Agents / biosynthesis*
  • Neurotransmitter Agents / metabolism
  • PC12 Cells
  • Rats

Substances

  • Butyrates
  • Neurotransmitter Agents
  • Calcium