Abstract
Scopoletin (1-50 microg/ml) inhibited the release of PGE2, TNF-alpha, IL-1beta and IL-6 and suppressed the expression of COX-2 in a concentration-dependent manner. These results suggest that scopoletin might suppress the production of such pro-inflammatory cytokines and exert inhibitory activity on LPS-induced PGE2 production through the depression of COX-2 expression.
Copyright 2004 Elsevier B.V.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Anti-Inflammatory Agents / administration & dosage
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Anti-Inflammatory Agents / pharmacology*
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Anti-Inflammatory Agents / therapeutic use
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Artemisia*
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Cell Line / drug effects
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Cyclooxygenase 2
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Dinoprostone / metabolism
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Dose-Response Relationship, Drug
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Interleukin-1 / metabolism
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Interleukin-6 / metabolism
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Isoenzymes / metabolism
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Lipopolysaccharides
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Macrophages / drug effects*
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Macrophages / metabolism*
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Mice
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Phytotherapy*
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Prostaglandin-Endoperoxide Synthases / metabolism
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Scopoletin / administration & dosage
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Scopoletin / pharmacology*
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Scopoletin / therapeutic use
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Tumor Necrosis Factor-alpha / metabolism
Substances
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Anti-Inflammatory Agents
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Interleukin-1
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Interleukin-6
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Isoenzymes
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Lipopolysaccharides
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Tumor Necrosis Factor-alpha
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Cyclooxygenase 2
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Prostaglandin-Endoperoxide Synthases
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Dinoprostone
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Scopoletin