[Effect of the two types of TNF-alpha on the biological functions of monocytic cell line U937]

Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2003 Jul;19(4):319-21.
[Article in Chinese]

Abstract

Aim: To compare the effect of the two types of TNF-alpha, transmembrane TNF-alpha (TM-TNF-alpha) and secreted TNF-alpha (sTNF-alpha) on the biological functions of monocytic cell line U937 so as to explore the role of TM-TNF-alpha in inflammation.

Methods: Effect of the two types of TNF-alpha on the functions of U937 cells, such as phagocytosis, accumulation of cytokines' mRNA, the level of cytoplasmic IkappaB-alpha and expression of ICAM-1, was compared by phagocytosis, RT-PCR, Western blot and FACS.

Results: sTNF-alpha was found to be able to promote markedly phagocytosis by monocytic U937 cells, enhance the mRNA accumulation of cytokines, such as TNF-alpha, IL-1beta and IL-8 mRNA, induce the degradation of IkappaB-alpha and the expression of adhesion molecule ICAM-1; while TM-TNF-alpha was shown to have no effect on the functions above of U937 cells. Furthermore, it seemed TM-TNF-alpha did not have a synergic action with sTNF-alpha when U937 cells was treated with both types of TNF-alpha.

Conclusion: The data above shows that sTNF-alpha can activate U937 cells, while TM-TNF-alpha has no influence. It suggests that the two types of TNF-alpha may play the different role in inflammation.

MeSH terms

  • Humans
  • Intercellular Adhesion Molecule-1
  • Interleukin-8 / metabolism
  • Monocytes* / metabolism
  • RNA, Messenger / metabolism
  • Tumor Necrosis Factor-alpha* / genetics
  • U937 Cells

Substances

  • Interleukin-8
  • RNA, Messenger
  • Tumor Necrosis Factor-alpha
  • Intercellular Adhesion Molecule-1