Subcortical Ca2+ waves sneaking under the plasma membrane in endothelial cells

Circ Res. 2004 Aug 6;95(3):e11-21. doi: 10.1161/01.RES.0000138447.81133.98. Epub 2004 Jul 8.

Abstract

Subplasmalemmal Ca2+, dynamically equilibrated with extracellular Ca2+, affects numerous signaling molecules, effectors, and events within this restricted space. We demonstrated the presence of a novel Ca2+ wave propagating beneath the plasma membrane in response to acute elevation of extracellular [Ca2+], by targeting a Ca2+ sensor, cameleon, to the endothelial plasmalemma. These subcortical waves, spatially distinct from classical cytosolic Ca2+ waves, originated in localized regions and propagated throughout the subplasmalemma. Translocation of an expressed GFP fused with a PH domain of PLC from the plasma membrane to the cytosol accompanied these subcortical waves, and U73122 attenuated not only the GFP-PH translocation, but also the peak amplitude of the subcortical Ca2+ waves; this finding suggests the involvement of local IP3 production through PLC-mediated PIP2 hydrolysis in the initiation of these waves. Changes in NO production as well as PKCbeta-GFP translocation from the cytosol to the plasma membrane, but not of GFP-PLA2 to perinuclear endomembranes, were associated with the subplasmalemmal Ca2+ changes. Thus, extracellular Ca2+ maintains the basal PLC activity of the plasma membrane, is involved in the initiation of compartmentalized subcortical Ca2+ waves, and regulates Ca2+-dependent signaling molecules residing in or translocated to the plasma membrane. The full text of this article is available online at http://circres.ahajournals.org.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Motifs
  • Calcium / metabolism
  • Calcium Signaling*
  • Calcium-Binding Proteins / analysis
  • Calcium-Binding Proteins / metabolism
  • Cell Compartmentation
  • Cell Membrane / physiology*
  • Cytosol / metabolism
  • Endothelial Cells / physiology*
  • Endothelial Cells / ultrastructure
  • Endothelium, Vascular / cytology*
  • Endothelium, Vascular / physiology
  • Estrenes / pharmacology
  • Extracellular Fluid / metabolism
  • Fluorescence Resonance Energy Transfer
  • Fluorescent Dyes / analysis
  • GAP-43 Protein / genetics
  • Genes, Reporter
  • Green Fluorescent Proteins / genetics
  • Ion Transport / physiology
  • Isoenzymes / genetics
  • Membrane Proteins / physiology*
  • Microscopy, Confocal
  • Nitric Oxide / metabolism
  • Phosphatidylinositol 4,5-Diphosphate / metabolism
  • Phospholipase C delta
  • Phospholipases A / genetics
  • Phospholipases A2
  • Protein Kinase C / genetics
  • Protein Kinase C / metabolism
  • Protein Kinase C beta
  • Protein Structure, Tertiary
  • Protein Transport
  • Pyrrolidinones / pharmacology
  • Recombinant Fusion Proteins / genetics
  • Recombinant Fusion Proteins / metabolism
  • Type C Phospholipases / genetics

Substances

  • Calcium-Binding Proteins
  • Estrenes
  • Fluorescent Dyes
  • GAP-43 Protein
  • Isoenzymes
  • Membrane Proteins
  • Phosphatidylinositol 4,5-Diphosphate
  • Pyrrolidinones
  • Recombinant Fusion Proteins
  • green fluorescent protein, Aequorea victoria
  • yellow cameleon
  • 1-(6-((3-methoxyestra-1,3,5(10)-trien-17-yl)amino)hexyl)-1H-pyrrole-2,5-dione
  • Green Fluorescent Proteins
  • Nitric Oxide
  • Protein Kinase C
  • Protein Kinase C beta
  • Phospholipases A
  • Phospholipases A2
  • Type C Phospholipases
  • Phospholipase C delta
  • Calcium