CD25(+)CD4(+) regulatory T cells from the peripheral blood of asymptomatic HIV-infected individuals regulate CD4(+) and CD8(+) HIV-specific T cell immune responses in vitro and are associated with favorable clinical markers of disease status

J Exp Med. 2004 Aug 2;200(3):331-43. doi: 10.1084/jem.20032069. Epub 2004 Jul 26.

Abstract

Human immunodeficiency virus (HIV) disease is associated with loss of CD4(+) T cells, chronic immune activation, and progressive immune dysfunction. HIV-specific responses, particularly those of CD4(+) T cells, become impaired early after infection, before the loss of responses directed against other antigens; the basis for this diminution has not been elucidated fully. The potential role of CD25(+)CD4(+) regulatory T cells (T reg cells), previously shown to inhibit immune responses directed against numerous pathogens, as suppressors of HIV-specific T cell responses was investigated. In the majority of healthy HIV-infected individuals, CD25(+)CD4(+) T cells significantly suppressed cellular proliferation and cytokine production by CD4(+) and CD8(+) T cells in response to HIV antigens/peptides in vitro; these effects were cell contact dependent and IL-10 and TGF-beta independent. Individuals with strong HIV-specific CD25(+) T reg cell function in vitro had significantly lower levels of plasma viremia and higher CD4(+): CD8(+) T cell ratios than did those individuals in whom this activity could not be detected. These in vitro data suggest that CD25(+)CD4(+) T reg cells may contribute to the diminution of HIV-specific T cell immune responses in vivo in the early stages of HIV disease.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • CD4 Antigens / analysis
  • CD4 Antigens / immunology*
  • CD8-Positive T-Lymphocytes / immunology*
  • Cytokines / biosynthesis
  • HIV / immunology*
  • HIV Infections / immunology*
  • Humans
  • Immune Tolerance
  • Interleukin-10 / physiology
  • Lymphocyte Activation
  • Receptors, Interleukin-2 / analysis*
  • T-Lymphocytes, Regulatory / immunology*
  • Transforming Growth Factor beta / physiology

Substances

  • CD4 Antigens
  • Cytokines
  • Receptors, Interleukin-2
  • Transforming Growth Factor beta
  • Interleukin-10