Effects of dietary long chain PUFAs on hippocampal lipid peroxidation and NMDA receptor subunits A and B concentration in streptozotocin-diabetic rats

Int J Neurosci. 2004 Oct;114(10):1353-64. doi: 10.1080/00207450490476147.

Abstract

This study examined the effects of streptozotocin (STZ)-diabetes and dietary long chain polyunsaturated fatty acids (LC-PUFAs) on hippocampal N-methyl-D-aspartate (NMDA) receptor subunit expression and lipid peroxidation. MDA level was significantly increased after 8 weeks of STZ-diabetes. LC-PUFAs administration significantly reduced MDA levels in diabetic rats. NR2A and NR2B protein concentrations were significantly decreased by about 30% in diabetic rats. Dietary LC-PUFAs partially restored NR2A and NR2B in diabetic rats whereas the most significant increase was seen in nondiabetic rats. Consequently, dietary LC-PUFAs can partially restore hippocampal NMDA receptors and decrease lipid peroxidation in diabetes. LC-PUFAs are thus a possible prophylactic means for preventing the cognitive deficiencies of diabetes.

Publication types

  • Comparative Study

MeSH terms

  • Animals
  • Blood Glucose / drug effects
  • Blotting, Western / methods
  • Body Weight / drug effects
  • Diabetes Mellitus, Experimental / metabolism*
  • Dietary Fats, Unsaturated / pharmacology
  • Fatty Acids, Unsaturated / pharmacology*
  • Hippocampus / drug effects*
  • Lipid Peroxidation / drug effects*
  • Malondialdehyde / metabolism
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, N-Methyl-D-Aspartate / drug effects
  • Receptors, N-Methyl-D-Aspartate / metabolism*
  • Statistics, Nonparametric

Substances

  • Blood Glucose
  • Dietary Fats, Unsaturated
  • Fatty Acids, Unsaturated
  • NR2A NMDA receptor
  • NR2B NMDA receptor
  • Receptors, N-Methyl-D-Aspartate
  • Malondialdehyde