CpG DNA induces IgG class switch DNA recombination by activating human B cells through an innate pathway that requires TLR9 and cooperates with IL-10

J Immunol. 2004 Oct 1;173(7):4479-91. doi: 10.4049/jimmunol.173.7.4479.

Abstract

TLRs are pattern recognition receptors that initiate innate immune responses. TLR9 detects microbial DNA with hypomethylated CpG motifs and in humans is preferentially expressed by IFN-alpha-producing plasmacytoid dendritic cells and B cells. In addition to favoring IFN-alpha release, TLR9 signals B cell activation, proliferation, and IgM production. Recent findings suggest that CpG DNA-TLR9 interaction plays a key role in systemic lupus erythematosus and rheumatoid arthritis, two autoimmune disorders characterized by dysregulated production of DNA-reactive IgG. We show that CpG DNA initiates germline C(gamma)1, C(gamma)2, and C(gamma)3 gene transcription by activating B cells through a TLR9-mediated NF-kappaB-Rel-dependent innate pathway that cooperates with IL-10 through STAT proteins and IFN-responsive factors. This pathway is inhibited by chloroquine, a drug that attenuates the clinical manifestations of IgG-mediated autoimmune disorders. Germline C(gamma) gene transcription is associated with up-regulation of activation-induced cytidine deaminase, a key element of the B cell class switch-inducing machinery, and is followed by class switch DNA recombination from C(micro) to C(gamma)1, C(gamma)2, and C(gamma)3. Subsequent IgG production requires additional signals from BCR and a B cell-activating factor of the TNF family (BAFF), produced by dendritic cells upon exposure to IFN-alpha. Our findings suggest that CpG DNA-TLR9 interaction may be important to initiate or amplify early T cell-independent IgG responses against pathogens. This implies that CpG DNA released during infections may exacerbate autoimmunity by stimulating autoreactive B cells to switch from an IgM to a more pathogenic IgG isotype.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adaptor Proteins, Signal Transducing
  • Adjuvants, Immunologic / physiology
  • Antigens, Differentiation / physiology
  • B-Cell Activating Factor
  • B-Lymphocytes / drug effects
  • B-Lymphocytes / enzymology
  • B-Lymphocytes / immunology
  • CD40 Ligand / physiology
  • Cell Line
  • Chloroquine / pharmacology
  • Cytidine Deaminase / biosynthesis
  • DNA, Bacterial / pharmacology*
  • DNA, Bacterial / physiology
  • Gene Rearrangement, B-Lymphocyte* / drug effects
  • Humans
  • Immunity, Innate / genetics
  • Immunoglobulin Class Switching / drug effects
  • Immunoglobulin Class Switching / genetics*
  • Immunoglobulin Constant Regions / genetics
  • Immunoglobulin Constant Regions / metabolism
  • Immunoglobulin G / biosynthesis*
  • Immunoglobulin G / genetics
  • Immunoglobulin G / metabolism
  • Immunoglobulin gamma-Chains / genetics
  • Immunoglobulin gamma-Chains / metabolism
  • Immunoglobulin mu-Chains / genetics
  • Immunoglobulin mu-Chains / metabolism
  • Interleukin-1 Receptor-Associated Kinases
  • Interleukin-10 / pharmacology
  • Interleukin-10 / physiology*
  • Lymphocyte Activation / genetics*
  • Membrane Glycoproteins / biosynthesis
  • Membrane Glycoproteins / physiology*
  • Membrane Proteins / physiology
  • Myeloid Differentiation Factor 88
  • NF-kappa B / physiology
  • NF-kappa B p50 Subunit
  • Promoter Regions, Genetic / immunology
  • Protein Kinases / physiology
  • Proteins / physiology
  • Receptors, Antigen, B-Cell / metabolism
  • Receptors, Antigen, B-Cell / physiology
  • Receptors, Cell Surface / biosynthesis
  • Receptors, Cell Surface / physiology*
  • Receptors, Immunologic / physiology
  • Response Elements / immunology
  • Signal Transduction / drug effects
  • Signal Transduction / genetics
  • Signal Transduction / immunology*
  • TNF Receptor-Associated Factor 6
  • Toll-Like Receptor 9
  • Toll-Like Receptors
  • Transcriptional Activation / drug effects
  • Transcriptional Activation / immunology
  • Tumor Necrosis Factor-alpha / physiology
  • Up-Regulation / immunology

Substances

  • Adaptor Proteins, Signal Transducing
  • Adjuvants, Immunologic
  • Antigens, Differentiation
  • B-Cell Activating Factor
  • CpG-DNA, E coli
  • DNA, Bacterial
  • Immunoglobulin Constant Regions
  • Immunoglobulin G
  • Immunoglobulin gamma-Chains
  • Immunoglobulin mu-Chains
  • MYD88 protein, human
  • Membrane Glycoproteins
  • Membrane Proteins
  • Myeloid Differentiation Factor 88
  • NF-kappa B
  • NF-kappa B p50 Subunit
  • Proteins
  • Receptors, Antigen, B-Cell
  • Receptors, Cell Surface
  • Receptors, Immunologic
  • TLR9 protein, human
  • TNF Receptor-Associated Factor 6
  • TNFSF13B protein, human
  • Toll-Like Receptor 9
  • Toll-Like Receptors
  • Tumor Necrosis Factor-alpha
  • Interleukin-10
  • CD40 Ligand
  • Chloroquine
  • Protein Kinases
  • Interleukin-1 Receptor-Associated Kinases
  • AICDA (activation-induced cytidine deaminase)
  • Cytidine Deaminase