Essential role of vascular endothelial growth factor and Flt-1 signals in neointimal formation after periadventitial injury

Arterioscler Thromb Vasc Biol. 2004 Dec;24(12):2284-9. doi: 10.1161/01.ATV.0000147161.42956.80. Epub 2004 Oct 7.

Abstract

Objective: Vascular endothelial growth factor (VEGF) is upregulated after arterial injury. Its role in the pathogenesis of neointimal formation after periadventitial injury, however, has not been addressed.

Methods and results: Expression of VEGF and its receptors but not that of placental growth factor markedly increased with the development of neointimal formation in hypercholesterolemic mice after cuff-induced periarterial injury. Transfection with the murine soluble Flt-1 (sFlt-1) gene to block VEGF in vivo in mice inhibited early inflammation and later neointimal formation. The sFlt-1 gene transfer did not affect plasma lipid levels but attenuated increased expression of VEGF, Flt-1, Flk-1, monocyte chemoattractant protein-1, and other inflammation-promoting factors. Mice with Flt-1 kinase deficiency also displayed reduced neointimal formation.

Conclusions: Inflammatory changes mediated by VEGF and Flt-1 signals play an important role in the pathogenesis of neointimal formation after cuff-induced periadventitial injury. VEGF might promote neointimal formation by acting as a proinflammatory cytokine.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apolipoproteins E / deficiency
  • Apolipoproteins E / physiology
  • Femoral Artery / chemistry
  • Femoral Artery / injuries
  • Femoral Artery / metabolism
  • Gene Expression Regulation / genetics
  • Gene Transfer Techniques
  • Hyperplasia / blood
  • Hyperplasia / enzymology
  • Immunohistochemistry / methods
  • Lipids / blood
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Neovascularization, Physiologic / physiology*
  • Protein Isoforms / genetics
  • Signal Transduction / physiology*
  • Solubility
  • Time Factors
  • Tunica Intima / pathology
  • Tunica Intima / physiology*
  • Vascular Endothelial Growth Factor Receptor-1 / deficiency
  • Vascular Endothelial Growth Factor Receptor-1 / genetics
  • Vascular Endothelial Growth Factor Receptor-1 / immunology
  • Vascular Endothelial Growth Factor Receptor-1 / metabolism*
  • Vascular Endothelial Growth Factor Receptor-2 / biosynthesis
  • Vascular Endothelial Growth Factor Receptor-2 / immunology
  • Vascular Endothelial Growth Factors / genetics

Substances

  • Apolipoproteins E
  • Lipids
  • Protein Isoforms
  • Vascular Endothelial Growth Factors
  • Vascular Endothelial Growth Factor Receptor-1
  • Vascular Endothelial Growth Factor Receptor-2