Protective effect of rhIL-1beta on pancreatic islets of alloxan-induced diabetic rats

World J Gastroenterol. 2004 Nov 15;10(22):3353-5. doi: 10.3748/wjg.v10.i22.3353.

Abstract

Aim: To observe the protective effect of rhIL-1beta on pancreatic islets of alloxan-induced diabetic rats.

Methods: Protection of rhIL-1beta on pancreatic islets of alloxan-induced diabetic rats (n = 5) was demonstrated with methods of immunohistochemistry and stereology. The concentration of serum glucose was measured by GOD method and that of serum insulin by RIA.

Results: The concentration of serum glucose increased but that of insulin decreased after administration of alloxan(150 mg/kg), and the volume density and numerical density of the islets were zero. In rhIL-1beta pretreated rats, although the concentration of serum insulin decreased (from 11.9+/-3.0 mIU/L to 6.1+/-1.6 mIU/L, P<0.05), that of glucose was at normal level compared with the control group. As compared with alloxan group, the concentration of serum glucose in rhIL-1beta pretreated rats decreased (from 19.4+/-8.9 mmol/L to 12.0+/-4.0 mmol/L, P<0.05) and the volume density increased(0/L to. 1/L, P<0.05).

Conclusion: rhIL-1beta pretreatment may have protective effect on the islets of alloxan-induced diabetic rats.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Blood Glucose
  • Cytoprotection
  • Diabetes Mellitus, Experimental / drug therapy*
  • Diabetes Mellitus, Experimental / pathology
  • Immunohistochemistry
  • Insulin / metabolism
  • Insulin Secretion
  • Interleukin-1 / pharmacology*
  • Islets of Langerhans / drug effects*
  • Islets of Langerhans / metabolism
  • Islets of Langerhans / pathology
  • Male
  • Rats
  • Rats, Sprague-Dawley
  • Recombinant Proteins / pharmacology

Substances

  • Blood Glucose
  • Insulin
  • Interleukin-1
  • Recombinant Proteins