Mouse myotomes pairs exhibit left-right asymmetric expression of MLC3F and alpha-skeletal actin

Dev Dyn. 2004 Dec;231(4):795-800. doi: 10.1002/dvdy.20176.

Abstract

Most muscle originates from the myotomal compartment of the somites, paired structures flanking the neural tube. Whereas vertebrate embryos show molecular and morphological asymmetry about the left-right body axis, somitic myogenesis is thought to occur symmetrically. Here, we provide the first evidence that myotome pairs are transiently left-right asymmetric, with higher expression of alpha-skeletal actin and myosin light chain 3F (MLC3F) on the left side between embryonic day 9.5-10.25. In iv mutants with situs inversus, the asymmetric expression of alpha-skeletal actin and MLC3F was inverted, showing that this process is regulated by global left-right axis cues, initiated before gastrulation. However, although left-sided identity is later maintained by Pitx2 genes, we found that Pitx2c null embryos have normal left-biased expression of alpha-skeletal actin and MLC3F. Myotome asymmetry, therefore, is downstream of the iv mutation but upstream of, or unrelated to, the Pitx2c pathway.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / genetics*
  • Animals
  • Embryonic Development / physiology*
  • Female
  • Gene Expression Regulation, Developmental
  • Homeobox Protein PITX2
  • Homeodomain Proteins / genetics
  • Mice
  • Mice, Inbred C57BL
  • Mice, Mutant Strains
  • Muscle, Skeletal / embryology*
  • Muscle, Skeletal / physiology
  • Myosin Light Chains / genetics*
  • Nuclear Proteins / genetics
  • Pregnancy
  • Situs Inversus / genetics
  • Situs Inversus / physiopathology*
  • Transcription Factors

Substances

  • Actins
  • Homeodomain Proteins
  • Myosin Light Chains
  • Nuclear Proteins
  • Transcription Factors