Characterization of corticosteroid redosing in an in vitro cell line model

Am J Obstet Gynecol. 2004 Oct;191(4):1403-8. doi: 10.1016/j.ajog.2004.06.049.

Abstract

Objective: The purpose of this study was to investigate dexamethasone redosing as function of time and dose.

Study design: We studied the effect of 48 hours' exposure to various concentrations of dexamethasone in a human pulmonary adenocarcinoma cell line (H-441). We measured the level of surfactant protein B (SP-B) mRNA by quantitative reverse transcription-PCR after initial dexamethasone exposure, and after redosing, 1 or 2 weeks later. Values are mean +/- SE for 5 experiments. Comparisons were made by Mann-Whitney and Kruskal-Wallis test with significance set at P < .05.

Results: Induction of SP-B mRNA was maximal within 48 hours of the initial dexamethasone exposure. Redosing with the same dexamethasone concentration resulted in levels more than double those initially observed. Redosing with dexamethasone concentration 10 times lower had an effect comparable to that of the initial, higher concentration.

Conclusion: Our results suggest a residual effect of the initial exposure that potentiates redosing, allowing significant dose reductions.

Publication types

  • Comparative Study

MeSH terms

  • Adenocarcinoma / pathology
  • Cell Line, Tumor / drug effects
  • Dexamethasone / administration & dosage*
  • Dose-Response Relationship, Drug
  • Gene Expression Regulation, Neoplastic / drug effects
  • Glucocorticoids / administration & dosage*
  • Humans
  • Lung
  • Lung Neoplasms / pathology
  • Polymerase Chain Reaction
  • Pulmonary Surfactant-Associated Protein B / drug effects*
  • Pulmonary Surfactant-Associated Protein B / genetics
  • RNA, Messenger / analysis
  • Time Factors
  • Tumor Cells, Cultured

Substances

  • Glucocorticoids
  • Pulmonary Surfactant-Associated Protein B
  • RNA, Messenger
  • Dexamethasone