Abstract
Maturity-onset diabetes of the young (MODY) is a rare subtype of type 2 diabetes that is characterized by autosomal-dominant inheritance and can be caused by mutations in hepatocyte nuclear factor 4alpha (HNF-4alpha). Odom and colleagues have combined chromatin immunoprecipitation with promoter microarrays to identify numerous promoters occupied by HNF-4alpha in the human liver and islet, suggesting a very broad role for HNF-4alpha in glucose homeostasis. This notion is supported by recent genetic studies linking HNF-4alpha to the much more common late-onset type 2 diabetes.
MeSH terms
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Adolescent
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Adult
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Age of Onset
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Blood Glucose / genetics
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Blood Glucose / metabolism
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Child
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Child, Preschool
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Chromatin Immunoprecipitation
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DNA-Binding Proteins / genetics
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DNA-Binding Proteins / physiology*
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Diabetes Mellitus, Type 2 / genetics*
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Diabetes Mellitus, Type 2 / metabolism
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Hepatocyte Nuclear Factor 4
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Hepatocytes / metabolism
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Homeostasis
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Humans
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Infant
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Islets of Langerhans / metabolism
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Phosphoproteins / genetics
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Phosphoproteins / physiology*
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Polymorphism, Single Nucleotide
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Promoter Regions, Genetic / genetics
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Transcription Factors / genetics
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Transcription Factors / physiology*
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Transcription, Genetic*
Substances
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Blood Glucose
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DNA-Binding Proteins
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HNF4A protein, human
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Hepatocyte Nuclear Factor 4
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Phosphoproteins
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Transcription Factors