Patterns of coordinate down-regulation of ARE-containing transcripts following immune cell activation

Genomics. 2004 Dec;84(6):1002-13. doi: 10.1016/j.ygeno.2004.08.007.

Abstract

We evaluated the expression of over 900 AU-rich element (ARE)-containing transcripts in primary human T lymphocytes following stimulation with anti-CD3 and anti-CD28 antibodies and found that approximately 48% of these transcripts were regulated following T cell activation. We identified approximately 145 ARE-containing transcripts that were rapidly induced and then rapidly disappeared within 1 h after activation. Another 250 ARE-containing transcripts expressed in resting T cells were rapidly turned off within 30 min after activation. The rates of transcript disappearance correlated well with rapid mRNA decay measured following transcriptional arrest with actinomycin D. We identified a subset of ARE-containing transcripts that were rapidly induced following T cell activation that were also induced following lipopolysaccharide stimulation of THP-1 monocytes, and these transcripts exhibited rapid decay in both cell types. Our results suggest that ARE-mediated mRNA decay plays an important role in the precisely coordinated down-regulation of gene expression following immune cell activation.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Blotting, Northern
  • CD28 Antigens / immunology
  • CD3 Complex / immunology
  • Dactinomycin / pharmacology
  • Down-Regulation
  • Gene Expression / drug effects
  • Humans
  • Lipopolysaccharides / pharmacology
  • Lymphocyte Activation / drug effects*
  • Monocytes / cytology
  • Monocytes / drug effects
  • Monocytes / metabolism
  • Oligonucleotide Array Sequence Analysis
  • Protein Synthesis Inhibitors / pharmacology
  • RNA Stability*
  • RNA, Messenger / drug effects
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism*
  • Regulatory Sequences, Nucleic Acid*
  • T-Lymphocytes / cytology
  • T-Lymphocytes / immunology
  • T-Lymphocytes / metabolism*
  • Transcription, Genetic*

Substances

  • CD28 Antigens
  • CD3 Complex
  • Lipopolysaccharides
  • Protein Synthesis Inhibitors
  • RNA, Messenger
  • Dactinomycin