[Pathophysiology and new treatment for hereditary neuropathy]

No To Shinkei. 2004 Sep;56(9):761-70.

[Article in Japanese]

Authors

Hiroyuki Nodera¹, Ryuji Kaji

Affiliation

¹ Department of Clinical Neuroscience, Graduate School of Medicine, the University of Tokushima, 2-50-1 Kuramotocho, Tokushima city, Tokushima 770-8503, Japan.

PMID: 15552865

No abstract available

Publication types

Review

MeSH terms

Animals
Ascorbic Acid / therapeutic use
Charcot-Marie-Tooth Disease / genetics*
Charcot-Marie-Tooth Disease / physiopathology*
Charcot-Marie-Tooth Disease / therapy
Connexins / genetics
Electrophysiology
Gap Junction beta-1 Protein
Gonanes / therapeutic use
Hormone Antagonists / therapeutic use
Humans
Mice
Myelin P0 Protein / genetics
Myelin Proteins / genetics*
Peripheral Nervous System / physiopathology
Potassium Channels / physiology
Proteins / genetics
Rats

Substances

CDRT1 protein, human
Connexins
Gonanes
Hormone Antagonists
Myelin P0 Protein
Myelin Proteins
PMP22 protein, human
Potassium Channels
Proteins
onapristone
Ascorbic Acid