Homotypic fibrillin-1 interactions in microfibril assembly

J Biol Chem. 2005 Feb 11;280(6):5013-21. doi: 10.1074/jbc.M409029200. Epub 2004 Nov 29.

Abstract

We have defined the homotypic interactions of fibrillin-1 to obtain new insights into microfibril assembly. Dose-dependent saturable high affinity binding was demonstrated between N-terminal fragments, between furin processed C-terminal fragments, and between these N- and C-terminal fragments. The N terminus also interacted with a downstream fragment. A post-furin cleavage site C-terminal sequence also interacted with the N terminus, with itself and with the furin-processed fragment. No other homotypic fibrillin-1 interactions were detected. Some terminal homotypic interactions were inhibited by other terminal sequences, and were strongly calcium-dependent. Treatment of an N-terminal fragment with N-ethylmaleimide reduced homotypic binding. Microfibril-associated glycoprotein-1 inhibited N- to C-terminal interactions but not homotypic N-terminal interactions. These fibrillin-1 interactions are likely to regulate pericellular fibrillin-1 microfibril assembly.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Motifs
  • Binding Sites
  • Calcium / metabolism
  • Contractile Proteins / chemistry
  • Dose-Response Relationship, Drug
  • Ethylmaleimide / chemistry
  • Extracellular Matrix Proteins / chemistry
  • Fibrillin-1
  • Fibrillins
  • Furin / chemistry
  • Humans
  • Kinetics
  • Ligands
  • Linear Models
  • Microfibrils / metabolism*
  • Microfilament Proteins / chemistry*
  • Microfilament Proteins / metabolism
  • Protein Binding
  • Protein Structure, Tertiary
  • RNA Splicing Factors
  • Recombinant Proteins / chemistry

Substances

  • Contractile Proteins
  • Extracellular Matrix Proteins
  • FBN1 protein, human
  • Fibrillin-1
  • Fibrillins
  • Ligands
  • Microfilament Proteins
  • RNA Splicing Factors
  • Recombinant Proteins
  • microfibrillar protein
  • Furin
  • Ethylmaleimide
  • Calcium