The genetic basis of the interaction between pyrimidine 5' nucleotidase I deficiency and hemoglobin E

Nucleosides Nucleotides Nucleic Acids. 2004 Oct;23(8-9):1261-3. doi: 10.1081/NCN-200027532.

Abstract

We have previously described a family in which the interaction between pyrimidine 5' nucleotidase I (P5N-I) deficiency and hemoglobin E resulted in severe haemolytic anaemia. In this study we explored the genetic basis of the severe clinical phenotype and look for evidence of the interaction between these conditions. A P5N-I gene mutation (IVS8 + 1-2delGT) was found in the family, confirming that the severe phenotype results from the interaction between two genetic diseases.

MeSH terms

  • 5'-Nucleotidase / deficiency*
  • 5'-Nucleotidase / genetics*
  • Anemia, Hemolytic / genetics
  • Exons
  • Family Health
  • Gene Deletion
  • Genotype
  • Hemoglobin E / genetics*
  • Hemoglobin E / metabolism*
  • Heterozygote
  • Homozygote
  • Humans
  • Mutation
  • Phenotype
  • Phosphoric Monoester Hydrolases / genetics
  • beta-Thalassemia / genetics

Substances

  • Hemoglobin E
  • uridine monophosphatase
  • Phosphoric Monoester Hydrolases
  • 5'-Nucleotidase