Age- and infection-related maturation of the nasal immune response in 0-2-year-old children

Allergy. 2005 Feb;60(2):226-32. doi: 10.1111/j.1398-9995.2005.00684.x.

Abstract

Background: The hygiene hypothesis suggests that exposure to micro-organisms influences development of the immune system in children.

Methods: In this study, we examined nasal immune responses in the first 2 years of life in relation to age of children and the number of viral infections they have experienced. Nasal brushes were taken during rhinovirus- (n = 20) or respiratory syncytial virus (RSV)-induced (n = 7) upper respiratory tract infections (URTI), and of controls (n = 40).

Results: The number of macrophages were higher during URTI and increased with age. The number of T lymphocytes increased with age in controls and were higher during URTI at all ages. We found an age-related decrease in the number of interleukin (IL)-4- and IL-10-positive cells in controls, while the number of IL-12-positive cells remained unchanged. Changes in T lymphocyte and IL-4 cell number were stronger related to the age of the child than to the number of respiratory infections, while the opposite was true for macrophages.

Conclusions: In infants, we found an infection- and age-related increase respectively for nasal macrophages and T lymphocytes during URTI. Furthermore, the number of IL-4- and IL-10-positive cells decreased with age. Whether this maturation reflects a natural age-related maturation, the degree of exposure to respiratory infections, or possibly both, could not be resolved and needs further study.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Age Distribution
  • Aging / immunology*
  • Aging / metabolism
  • Antibody Formation
  • Case-Control Studies
  • Cohort Studies
  • Cytokines / metabolism
  • Female
  • Humans
  • Immune System / growth & development
  • Infant
  • Infant, Newborn
  • Infections
  • Macrophages / pathology
  • Male
  • Nasal Cavity / immunology*
  • Nasal Cavity / pathology
  • Picornaviridae Infections / epidemiology
  • Picornaviridae Infections / immunology*
  • Picornaviridae Infections / pathology
  • Respiratory Syncytial Virus Infections / epidemiology
  • Respiratory Syncytial Virus Infections / immunology*
  • Respiratory Syncytial Virus Infections / pathology
  • Respiratory Tract Infections / epidemiology
  • Respiratory Tract Infections / immunology*
  • Respiratory Tract Infections / pathology
  • Rhinovirus*
  • T-Lymphocytes / pathology

Substances

  • Cytokines