Role of Krüppel-like factor 15 in PEPCK gene expression in the liver

Biochem Biophys Res Commun. 2005 Feb 18;327(3):920-6. doi: 10.1016/j.bbrc.2004.12.096.

Abstract

Regulation of hepatic gene expression is important for energy homeostasis. We now show that hepatic expression of the gene for the transcription factor Kruppel-like factor 15 (KLF15) is increased by food deprivation and reduced by feeding in mice. Expression of the KLF15 gene in mouse liver was also down-regulated by a euglycemic-hyperinsulinemic clamp and was increased by inhibition of phosphatidylinositol 3-kinase. In cultured rat hepatocytes, KLF15 gene expression was induced by dexamethasone and a non-hydrolyzing analog of cAMP, and this effect was inhibited by insulin in a manner dependent on phosphatidylinositol 3-kinase signaling. Forced expression of KLF15 in cultured hepatocytes increased both the expression and the promoter activity of the gene for phosphoenolpyruvate carboxykinase (PEPCK). These results suggest that insulin and its counteracting hormones regulate the hepatic expression of KLF15, and that this transcription factor contributes to the regulation of PEPCK gene expression in the liver.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cells, Cultured
  • Cyclic AMP / analogs & derivatives
  • Cyclic AMP / pharmacology
  • DNA-Binding Proteins
  • Dexamethasone / pharmacology
  • Down-Regulation
  • Electrophoresis, Polyacrylamide Gel
  • Gene Expression Regulation, Enzymologic*
  • Glucose Clamp Technique / methods
  • Hepatocytes / drug effects
  • Hepatocytes / metabolism
  • Insulin / metabolism
  • Insulin / pharmacology
  • Kruppel-Like Transcription Factors
  • Liver / metabolism*
  • Mice
  • Phosphatidylinositol 3-Kinases / metabolism
  • Phosphoenolpyruvate Carboxykinase (GTP) / genetics
  • Phosphoenolpyruvate Carboxykinase (GTP) / metabolism*
  • Phosphoinositide-3 Kinase Inhibitors
  • Rats
  • Transcription Factors / chemistry
  • Transcription Factors / physiology*

Substances

  • DNA-Binding Proteins
  • Insulin
  • Klf15 protein, mouse
  • Kruppel-Like Transcription Factors
  • Phosphoinositide-3 Kinase Inhibitors
  • Transcription Factors
  • Dexamethasone
  • Cyclic AMP
  • Phosphoenolpyruvate Carboxykinase (GTP)