53BP2 induces apoptosis through the mitochondrial death pathway

Genes Cells. 2005 Mar;10(3):253-60. doi: 10.1111/j.1365-2443.2005.00835.x.

Abstract

The p53 binding protein 2 (53BP2) has been identified as the interacting protein to p53, Bcl-2, and p65 subunit of nuclear factor kappaB (NF-kappaB). The TP53BP2 gene encodes two splicing variants, 53BP2S and 53BP2L, previously known as apoptosis stimulating protein 2 of p53 (ASPP2). We found that these 53BP2 proteins are located predominantly in the cytoplasm and induce apoptosis as demonstrated by cleavage of poly ADP ribose polymerase (PARP) and annexin V staining. Furthermore, we demonstrate that 53BP2 is located in the mitochondria and induces apoptosis associated with depression of the mitochondrial trans-membrane potential (DeltaPsim) and activation of caspase-9. From these findings we conclude that 53BP2 induces apoptosis through the mitochondrial death pathway.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / physiology*
  • Apoptosis Regulatory Proteins
  • Carrier Proteins / metabolism*
  • Caspase 9
  • Caspases / metabolism
  • Cytosol / metabolism
  • Ecdysterone / analogs & derivatives*
  • Ecdysterone / metabolism
  • Humans
  • Mitochondria / physiology*

Substances

  • Apoptosis Regulatory Proteins
  • Carrier Proteins
  • TP53BP2 protein, human
  • Ecdysterone
  • ponasterone A
  • CASP9 protein, human
  • Caspase 9
  • Caspases