Cycling G1 CD34+/CD38+ cells potentiate the motility and engraftment of quiescent G0 CD34+/CD38-/low severe combined immunodeficiency repopulating cells

Stem Cells. 2005 Apr;23(4):561-74. doi: 10.1634/stemcells.2004-0060.

Abstract

The mechanism of human stem cell expansion ex vivo is not fully understood. Furthermore, little is known about the mechanisms of human stem cell homing/repopulation and the role that differentiating progenitor cells may play in these processes. We report that 2- to 3-day in vitro cytokine stimulation of human cord blood CD34(+)-enriched cells induces the production of short-term repopulating, cycling G1 CD34(+)/CD38(+) cells with increased matrix metalloproteinase (MMP)-9 secretion as well as increased migration capacity to the chemokine stromal cell-derived factor-1 (SDF-1) and homing to the bone marrow of irradiated nonobese diabetic severe/combined immunodeficiency (NOD/SCID) mice. These cycling G1 cells enhance SDF-1-mediated in vitro migration and in vivo homing of quiescent G0 CD34(+) cells, which is partially abrogated after inhibition of MMP-2/-9 activity. Moreover, the engraftment potential of quiescent G0 SCID repopulating cells (SRCs) is also increased by the cycling G1 CD34(+)/CD38(+) cells. This effect is significantly abrogated after incubation of cycling G1 cells with a neutralizing anti-CXCR4 antibody. Our data suggest synergistic interactions between accessory cycling G1 CD34(+)/CD38(+) committed progenitor cells and quiescent, primitive G0 CD34(+)/CD38(-/low) SRC/stem cells, the former increasing the motility and engraftment potential of the latter, partly via secretion of MMP-9.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • ADP-ribosyl Cyclase 1 / metabolism*
  • Animals
  • Antigens, CD34 / metabolism*
  • Bone Marrow Cells / physiology
  • Cells, Cultured
  • Chemokine CXCL12
  • Chemokines, CXC / metabolism
  • Chemotaxis
  • Colony-Forming Units Assay
  • Cytokines / metabolism
  • Fetal Blood / cytology
  • G1 Phase*
  • Humans
  • Matrix Metalloproteinase 9 / metabolism
  • Mice
  • Mice, Inbred NOD
  • Mice, SCID
  • Receptors, CXCR4 / metabolism
  • Resting Phase, Cell Cycle*
  • Stem Cell Transplantation
  • Stem Cells / cytology*
  • Stem Cells / physiology

Substances

  • Antigens, CD34
  • CXCL12 protein, human
  • Chemokine CXCL12
  • Chemokines, CXC
  • Cxcl12 protein, mouse
  • Cytokines
  • Receptors, CXCR4
  • ADP-ribosyl Cyclase 1
  • Matrix Metalloproteinase 9