Variation in ITGB3 is associated with asthma and sensitization to mold allergen in four populations

Am J Respir Crit Care Med. 2005 Jul 1;172(1):67-73. doi: 10.1164/rccm.200411-1555OC. Epub 2005 Apr 7.

Abstract

Rationale: Recent genetic studies have implicated integrins in asthma and atopy susceptibility. We therefore evaluated the integrin-beta3 gene (ITGB3), an integrin gene within an asthma linkage peak on chromosome 17, as a candidate for susceptibility to asthma- and atopy-related phenotypes.

Methods and measurements: We genotyped and performed association tests on 19 single nucleotide polymorphisms in ITGB3 in the Hutterites, a founder population, and in three outbred replication populations.

Main results: Variation in ITGB3 was strongly associated with susceptibility to bronchial hyperresponsiveness and protection from allergic sensitization to mold allergens in this population. Three independent case-control populations representing Caucasians and African Americans were used to replicate this finding, also revealing ITGB3 alleles that are associated with asthma susceptibility and protection from mold allergen sensitization.

Conclusions: This study provides evidence that ITGB3 plays a role in the pathogenesis of asthma and sensitization to mold allergens.

Publication types

  • Multicenter Study
  • Research Support, N.I.H., Extramural
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Allergens / adverse effects*
  • Antigens, Dermatophagoides / adverse effects*
  • Asthma / genetics*
  • Black or African American / genetics
  • Case-Control Studies
  • Chromosomes, Human, Pair 17 / genetics
  • Genetic Predisposition to Disease
  • Genetic Variation
  • Humans
  • Integrin beta3 / genetics*
  • Polymorphism, Single Nucleotide*
  • Respiratory Hypersensitivity / genetics
  • Statistics, Nonparametric
  • White People / genetics

Substances

  • Allergens
  • Antigens, Dermatophagoides
  • ITGB3 protein, human
  • Integrin beta3