Complementary contribution of CD4 and CD8 T lymphocytes to T-cell infiltration of the intact and the degenerative spinal cord

Am J Pathol. 2005 May;166(5):1441-50. doi: 10.1016/S0002-9440(10)62361-9.

Abstract

The central role of T cells in inflammatory reactions of the central nervous system (CNS) is well documented. However, there is little information about the few T cells found within the noninflamed CNS. In particular, the contribution of CD4+ and CD8+ T cells to the lymphocyte pool infiltrating the intact CNS, the location of these cells in CNS white and gray matter, and changes in the cellular composition of T-cell infiltrates coinciding with degeneration are primarily undefined. To address these points, we studied T cells in the intact and degenerative rat spinal cord. In the intact spinal cord, T cells were preferentially located within the gray matter. CD8+ T cells were more numerous than CD4+ lymphocytes. In cases of neuroaxonal degeneration or myelin degeneration/oligodendrocyte death, T cells were predominantly seen in areas of degeneration and were present in increased numbers. These effects were more pronounced for the CD4+ than for the CD8+ T-cell subset. Collectively, these data provide evidence for a clear cellular and compartmental bias in T-cell infiltration of the intact and degenerative spinal cord. This could indicate that CD4+ and CD8+ T cells might fulfill complementary roles in the intact and the diseased organ.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aging
  • Animals
  • Animals, Genetically Modified
  • Axons / pathology
  • CD4-Positive T-Lymphocytes / pathology*
  • CD8-Positive T-Lymphocytes / pathology*
  • Lymphocyte Count
  • Myelin Proteolipid Protein / metabolism
  • Myelin Sheath / pathology
  • Nerve Degeneration / metabolism
  • Nerve Degeneration / pathology*
  • Nerve Tissue Proteins / metabolism
  • Rats
  • Rats, Inbred Lew
  • Spinal Cord / metabolism
  • Spinal Cord / pathology
  • Spinal Cord Diseases / metabolism
  • Spinal Cord Diseases / pathology*

Substances

  • Myelin Proteolipid Protein
  • Nerve Tissue Proteins
  • Plp1 protein, rat