The antibodies directed against N-terminal heptad-repeat peptide of hRSV fusion protein and its analog-5-Helix inhibit virus infection in vitro

Biochem Biophys Res Commun. 2005 Jun 17;331(4):1358-64. doi: 10.1016/j.bbrc.2005.04.046.

Abstract

Human respiratory syncytial virus (hRSV) membrane fusion is promoted by the formation of a trimer-of-hairpins structure that brings the amino- and carboxyl-terminal regions of fusion (F) protein into close proximity. Two heptad-repeat (HR1 and HR2) regions in F protein play an important role in this process. Our previous study demonstrated that peptides derived from HR1 and HR2 regions of F protein were potent inhibitors of hRSV entry. Here we showed that HR1 peptide and its analog denoted 5-Helix which contained a central coiled-coil formed by three HR1s could induce highly potent antibody response in the immunized rabbits. Both antibodies could recognize F1 domain of the F protein and inhibited hRSV entry with the neutralizing antibody titers of 1:61 and 1:115, respectively. These suggested that 5-Helix could induce potent neutralizing antibody response and the central coiled-coil might be a highly conserved neutralization site for hRSV F protein.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Antibodies / immunology*
  • Antiviral Agents / physiology*
  • Cell Line
  • Electrophoresis, Polyacrylamide Gel
  • Immune Sera
  • Molecular Sequence Data
  • Neutralization Tests
  • Peptide Fragments / immunology*
  • Respiratory Syncytial Viruses / metabolism*
  • Viral Fusion Proteins / chemistry*

Substances

  • Antibodies
  • Antiviral Agents
  • Immune Sera
  • Peptide Fragments
  • Viral Fusion Proteins