Abstract
Insulin and cAMP have opposing effects on de novo fatty acid synthesis in liver and in cultured hepatocytes mediated by sterol-regulatory element binding protein (SREBP). To determine whether these agents regulate the cleavage of full-length SREBP to generate the transcriptionally active N-terminal fragment (nSREBP) in primary rat hepatocytes, an adenoviral vector (Ad-SREBP-1a) was constructed to constitutively express full-length SREBP-1a. Insulin increased, and dibutyryl (db)-cAMP inhibited, generation of nSREBP-1a from its full-length precursor. Insulin stimulated processing of SREBP-1a within 1h, and the effect was sustained for at least 24h. The initial stimulation of SREBP processing by insulin preceded measurable reduction in Insig-2 mRNA levels. Rat hepatocytes were also infected with an adenovirus expressing the nuclear form of SREBP-1c (Ad-nSREBP-1c). Insulin increased the half-life of constitutively expressed nSREBP-1c, and this effect of insulin was also inhibited by db-cAMP.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, Non-P.H.S.
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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Animals
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CCAAT-Enhancer-Binding Proteins / metabolism*
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Cells, Cultured
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Cyclic AMP / metabolism*
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DNA-Binding Proteins / metabolism*
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Dose-Response Relationship, Drug
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Hepatocytes / drug effects
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Hepatocytes / metabolism*
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Insulin / metabolism*
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Insulin / pharmacology
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Intracellular Signaling Peptides and Proteins / metabolism*
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Male
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Membrane Proteins / metabolism*
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Protein Processing, Post-Translational / drug effects
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Protein Processing, Post-Translational / physiology*
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Rats
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Rats, Sprague-Dawley
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Sterol Regulatory Element Binding Protein 1
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Transcription Factors / metabolism*
Substances
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CCAAT-Enhancer-Binding Proteins
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DNA-Binding Proteins
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INSIG2 protein, human
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Insulin
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Intracellular Signaling Peptides and Proteins
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Membrane Proteins
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Srebf1 protein, rat
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Sterol Regulatory Element Binding Protein 1
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Transcription Factors
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Cyclic AMP