Failure of d-cycloserine to reverse cognitive disruption induced by scopolamine or phencyclidine in primates

Life Sci. 1992;50(25):1959-62. doi: 10.1016/0024-3205(92)90525-t.

Abstract

The partial glycine agonist d-cycloserine may be of therapeutic use as a cognitive enhancer. We examined the ability of d-cycloserine (3-14 mg/kg i.m.) to reverse cognitive impairments induced by scopolamine (0.03 mg/kg) or PCP (0.25 mg/kg). There was no evidence for a dose-related improvement in visuospatial memory using retention intervals of 2,10 or 20 sec. A possible explanation for these findings is that central glycine function cannot be adequately increased using systemic administration of d-cycloserine in this dose range.

MeSH terms

  • Animals
  • Cycloserine / administration & dosage
  • Cycloserine / pharmacology*
  • Macaca mulatta
  • Male
  • Memory / drug effects*
  • Phencyclidine / pharmacology*
  • Random Allocation
  • Scopolamine / pharmacology*

Substances

  • Cycloserine
  • Scopolamine
  • Phencyclidine