Transforming growth factor beta1-modified donor cell transfusion induced allo-heart tolerance

Transplant Proc. 2005 Jun;37(5):2360-4. doi: 10.1016/j.transproceed.2005.03.106.

Abstract

The therapeutic value of the transforming growth factor beta 1 (TGF-beta1) in transplantation has been reported; however, cell-mediated gene therapy using TGF-beta1 is not a widespread application in organ transplantation. This study was performed to evaluate whether TGF-beta1-modified donor spleen cell-specific transfusion could induce tolerogenicity and prolong allograft survival in rat heterotopic heart transplantation. Stable TGF-beta1-transduced spleen cells were established. Wistar rat splenic T-cell responses to donor spleen cells that received TGF-beta1-transduced were severely impaired. Survival of Sprague-Dawley cardiac allografts in Wistar rats given TGF-beta1-modified donor spleen cells (5 x 10(6), 7 days before transplantation), was extended modestly but significantly. Liposome transduction of donor spleen cells to overexpress TGF-beta1 is associated with marked impairment of their T-cell allostimulatory activity but only modest prolongation of allo-heart survival.

MeSH terms

  • Animals
  • Gene Transfer Techniques
  • Graft Survival / physiology*
  • Growth Substances / genetics
  • Heart Transplantation / immunology*
  • Heart Transplantation / pathology
  • Immune Tolerance
  • Lymphocyte Activation
  • Lymphocyte Culture Test, Mixed
  • Lymphocyte Transfusion
  • Models, Animal
  • Rats
  • Rats, Inbred WF
  • Rats, Sprague-Dawley
  • Spleen / cytology
  • Spleen / immunology
  • T-Lymphocytes / immunology
  • Transforming Growth Factor beta / genetics*
  • Transforming Growth Factor beta / immunology*
  • Transforming Growth Factor beta1
  • Transplantation, Heterologous

Substances

  • Growth Substances
  • Tgfb1 protein, rat
  • Transforming Growth Factor beta
  • Transforming Growth Factor beta1