A possible mechanism for atherosclerosis induced by polycyclic aromatic hydrocarbons

Biochem Biophys Res Commun. 2005 Sep 16;335(1):220-6. doi: 10.1016/j.bbrc.2005.07.062.

Abstract

Polycyclic aromatic hydrocarbons (PAHs), aryl hydrocarbon receptor (AHR) ligands, induce atherogenesis. Liver X receptor (LXR) alpha is known to be involved in the control of cholesterol homeostasis. Thus, the purpose of this study was to investigate the effects of 3-methlycholanthrene (MC), one of the PAHs, on LXRalpha-mediated signal transductions. We found that expression of mRNAs for ATP binding cassette A1, sterol regulatory element binding protein 1c (SREBP-1c), fatty acid synthase, and stearoyl-CoA desaturase was suppressed by treatment of HepG2 cells with MC. A luciferase reporter assay revealed that LXRalpha- and SREBP-1c-mediated transactivations were inhibited by MC via AHR. Based on these lines of evidence, we propose that down-regulation of the LXRalpha-regulated genes by PAHs is one of the causes responsible for atherosclerosis induced by PAHs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Arteriosclerosis / chemically induced*
  • Arteriosclerosis / genetics
  • Arteriosclerosis / metabolism
  • Benz(a)Anthracenes / pharmacology
  • Benz(a)Anthracenes / toxicity
  • CCAAT-Enhancer-Binding Proteins / genetics
  • CCAAT-Enhancer-Binding Proteins / metabolism
  • Cell Line, Tumor
  • DNA-Binding Proteins / genetics
  • DNA-Binding Proteins / metabolism
  • Gene Expression Regulation / drug effects
  • Humans
  • Liver X Receptors
  • Models, Biological
  • Orphan Nuclear Receptors
  • Polycyclic Aromatic Hydrocarbons / pharmacology*
  • Polycyclic Aromatic Hydrocarbons / toxicity
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • RNA, Small Interfering / genetics
  • Receptors, Aryl Hydrocarbon / genetics
  • Receptors, Aryl Hydrocarbon / metabolism
  • Receptors, Cytoplasmic and Nuclear / genetics
  • Sterol Regulatory Element Binding Protein 1
  • Transcription Factors / genetics
  • Transcription Factors / metabolism
  • Transcription, Genetic / drug effects
  • Transcription, Genetic / genetics

Substances

  • 3-methlycholanthrene
  • Benz(a)Anthracenes
  • CCAAT-Enhancer-Binding Proteins
  • DNA-Binding Proteins
  • Liver X Receptors
  • NR1H3 protein, human
  • Orphan Nuclear Receptors
  • Polycyclic Aromatic Hydrocarbons
  • RNA, Messenger
  • RNA, Small Interfering
  • Receptors, Aryl Hydrocarbon
  • Receptors, Cytoplasmic and Nuclear
  • SREBF1 protein, human
  • Sterol Regulatory Element Binding Protein 1
  • Transcription Factors