YM-201627: an orally active antitumor agent with selective inhibition of vascular endothelial cell proliferation

Cancer Lett. 2006 Jul 8;238(1):119-27. doi: 10.1016/j.canlet.2005.06.037. Epub 2005 Aug 10.

Abstract

We developed an oral administration-compatible, small molecular weight antitumor agent, YM-201627 by screening for the inhibition of the proliferation of VEGF-stimulated HUVECs. YM-201627 selectively inhibited the proliferation of various endothelial cell lines induced by VEGF, bFGF, and FBS (at IC50s of 0.0039-0.12 microM), that would not be expected to have any direct antiproliferative effect on other cell types. YM-201627 inhibited angiogenesis in vitro at a concentration of 0.01 microM. In the in vivo studies, it inhibited microvessel formation induced by human melanoma A375 cells suspended in Matrigel (86% with twice-daily doses of 30 mg/kg). Moreover, once-daily oral dosing of YM-201627 to mice bearing A375 xenografts elicited significant antitumor activity (73% with daily doses of 10 mg/kg). These results suggest that YM-201627 is a selective growth inhibitor of endothelial cells, which may be useful for treatment of solid tumors.

MeSH terms

  • Administration, Oral
  • Angiogenesis Inducing Agents / pharmacology
  • Animals
  • Antineoplastic Agents / therapeutic use*
  • Cell Line, Tumor
  • Cell Proliferation / drug effects*
  • Drug Screening Assays, Antitumor
  • Endothelial Cells / drug effects*
  • Humans
  • Imidazoles / chemistry
  • Imidazoles / therapeutic use*
  • Melanoma, Experimental / drug therapy*
  • Mice
  • Mice, Inbred Strains
  • Phosphorylation / drug effects
  • Vascular Endothelial Growth Factor Receptor-2 / analysis
  • Vascular Endothelial Growth Factor Receptor-2 / drug effects
  • Xenograft Model Antitumor Assays

Substances

  • Angiogenesis Inducing Agents
  • Antineoplastic Agents
  • Imidazoles
  • YM-201627
  • Vascular Endothelial Growth Factor Receptor-2