Zoledronate synergises with imatinib mesylate to inhibit Ph primary leukaemic cell growth

Br J Haematol. 2005 Aug;130(4):558-60. doi: 10.1111/j.1365-2141.2005.05648.x.

Abstract

We examined the in vivo effects and safety of the third generation bisphosphonate, zoledronate (ZOL) alone and combined with imatinib mesylate against primary Philadelphia chromosome positive (Ph+) leukaemic cells. ZOL inhibited the prenylation of Rap1A in leukaemic cells in vitro and synergised with imatinib to enhance the survival of mice engrafted with cells from imatinib-responders, but not from non-responders because of mutated BCR-ABL. These findings suggest that the combination of ZOL and imatinib accelerate the eradication of Ph+ clone, resulting in better prognosis of Ph+ leukaemia patients who have not yet acquired mutations.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antineoplastic Combined Chemotherapy Protocols
  • Benzamides
  • Diphosphonates
  • Drug Synergism
  • Humans
  • Imatinib Mesylate
  • Imidazoles
  • Leukemia, Myelogenous, Chronic, BCR-ABL Positive / drug therapy*
  • Mice
  • Mice, SCID
  • Models, Animal
  • Neoplasm Transplantation
  • Piperazines
  • Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy*
  • Pyrimidines
  • Tumor Cells, Cultured
  • Zoledronic Acid

Substances

  • Benzamides
  • Diphosphonates
  • Imidazoles
  • Piperazines
  • Pyrimidines
  • Zoledronic Acid
  • Imatinib Mesylate