Emerging roles of Polycomb silencing in X-inactivation and stem cell maintenance

Cold Spring Harb Symp Quant Biol. 2004:69:319-26. doi: 10.1101/sqb.2004.69.319.
No abstract available

Publication types

  • Review

MeSH terms

  • Animals
  • DNA-Binding Proteins / genetics
  • DNA-Binding Proteins / metabolism
  • Dosage Compensation, Genetic*
  • Drosophila / genetics
  • Drosophila / metabolism
  • Drosophila Proteins / genetics*
  • Drosophila Proteins / metabolism
  • Epigenesis, Genetic
  • Female
  • Gene Silencing*
  • Histone-Lysine N-Methyltransferase
  • Histones / chemistry
  • Histones / metabolism
  • Humans
  • Male
  • Models, Genetic
  • Myeloid-Lymphoid Leukemia Protein
  • Neoplasms / etiology
  • Nucleosomes / metabolism
  • Polycomb Repressive Complex 1
  • Polycomb-Group Proteins
  • Proto-Oncogenes / genetics
  • Repressor Proteins / genetics
  • Repressor Proteins / metabolism
  • Stem Cells / metabolism*
  • Transcription Factors / genetics
  • Transcription Factors / metabolism
  • Ubiquitin / metabolism

Substances

  • DNA-Binding Proteins
  • Drosophila Proteins
  • Histones
  • KMT2A protein, human
  • Nucleosomes
  • Pc protein, Drosophila
  • Polycomb-Group Proteins
  • Repressor Proteins
  • Transcription Factors
  • Trl protein, Drosophila
  • Ubiquitin
  • Myeloid-Lymphoid Leukemia Protein
  • Histone-Lysine N-Methyltransferase
  • Polycomb Repressive Complex 1