Presenilin 1 Glu318Gly polymorphism: interpret with caution

Arch Neurol. 2005 Oct;62(10):1624-7. doi: 10.1001/archneur.62.10.1624.

Abstract

Background: The significance of the presenilin 1 (PSEN1) Glu318Gly polymorphism has been described as either a causal mutation with reduced penetrance or a benign polymorphism. When this polymorphism is found in a symptomatic person with a family history of dementia, counseling on recurrence risk becomes very problematic.

Objective: To demonstrate that the PSEN1 Glu318Gly polymorphism should be interpreted cautiously.

Design: Case histories of 2 patients with presenile dementia and family histories of dementia are described. The PSEN1 gene was sequenced in the patients and in 11 family members of patient 1.

Results: Two patients with presenile dementia and personality change were found to carry the PSEN1 Glu318Gly polymorphism. The presence of the polymorphism was confirmed in several family members of patient 1 but was absent in 1 symptomatic relative.

Conclusions: The Glu318Gly polymorphism may be associated with risk for neurodegenerative disease; however, in the cases described here, it did not appear to be a risk factor. Until there is consensus on whether it is associated with disease, families should be informed that the clinical significance of the polymorphism is uncertain.

Publication types

  • Case Reports
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Alzheimer Disease / genetics*
  • Alzheimer Disease / physiopathology
  • DNA Mutational Analysis
  • Female
  • Genetic Counseling
  • Genetic Predisposition to Disease*
  • Humans
  • Male
  • Membrane Proteins / genetics*
  • Middle Aged
  • Pedigree
  • Polymorphism, Genetic*
  • Presenilin-1
  • Risk Factors

Substances

  • Membrane Proteins
  • PSEN1 protein, human
  • Presenilin-1