Measures of oxidized low-density lipoprotein and oxidative stress are not related and not elevated in otherwise healthy men with the metabolic syndrome

Arterioscler Thromb Vasc Biol. 2005 Dec;25(12):2580-6. doi: 10.1161/01.ATV.0000190675.08857.3d. Epub 2005 Oct 13.

Abstract

Objective: The metabolic syndrome predisposes to the development of cardiovascular diseases. Oxidative stress and elevated circulating oxidized low-density lipoprotein (LDL) concentrations are related to cardiovascular disease and proposed to be features of the metabolic syndrome. F2-isoprostanes are lipid peroxidation products and considered the most reliable biomarkers of oxidative stress.

Methods and results: Plasma oxidized LDL (oxLDL) and urinary 8-iso-prostaglandin F2alpha (8-iso-PGF2alpha; the major F2-isoprostane) were analyzed in a cross-sectional study of 289 healthy men (62 to 64 years of age). Individuals completed a 7-day dietary record, and fasting plasma insulin, lipid, and lipoprotein concentrations, LDL particle size, and inflammatory markers were determined. National Cholesterol Education Program/Adult Treatment Panel III (NCEP/ATPIII) criteria were used to define the metabolic syndrome and individuals were grouped according to the number of risk factors for the metabolic syndrome (0, [n=88; 30%]; > or =1, [n=179; 62%], metabolic syndrome [n=22; 8%]). Group comparisons revealed no differences for oxLDL, 8-iso-PGF2alpha, or reported intake of macronutrients, whereas C-reactive protein and interleukin-6 were increased in the metabolic syndrome. LDL cholesterol strongly determined oxLDL in univariate and multivariate analysis, but no relationship to 8-iso-PGF2alpha was found. In turn, 8-iso-PGF2alpha was related to reported intake of fat, fatty acids, and dietary antioxidants.

Conclusions: There were no increases in plasma oxLDL or measures of oxidative stress (urinary 8-iso-PGF2alpha) in these otherwise healthy 63-year-old men with the metabolic syndrome. Furthermore, no relationship between oxLDL and 8-iso-PGF2alpha was found, but our results suggest a role for dietary factors in oxidative stress.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Atherosclerosis / diet therapy
  • Atherosclerosis / epidemiology
  • Atherosclerosis / metabolism*
  • Dinoprost / analogs & derivatives
  • Dinoprost / urine
  • Feeding Behavior
  • Humans
  • Lipoproteins, LDL / blood*
  • Male
  • Metabolic Syndrome / diet therapy
  • Metabolic Syndrome / epidemiology
  • Metabolic Syndrome / metabolism*
  • Middle Aged
  • Multivariate Analysis
  • Nutrition Assessment
  • Oxidative Stress / physiology*
  • Risk Factors

Substances

  • Lipoproteins, LDL
  • oxidized low density lipoprotein
  • 8-epi-prostaglandin F2alpha
  • Dinoprost