Beneficial effect of transfusion with low-affinity red blood cells in endotoxemia

Transfusion. 2005 Nov;45(11):1785-90. doi: 10.1111/j.1537-2995.2005.00603.x.

Abstract

Background: Sepsis caused by endotoxins such as lipopolysaccharide (LPS) impairs the microcirculation, diminishing tissue blood supply and aggravates systemic hypoxia. A novel lower-affinity hemoglobin (Hb) variant, Hb Presbyterian, enhances oxygen release to peripheral tissues and may improve tissue oxygen supply during sepsis.

Study design and methods: This study investigated the effectiveness of Presbyterian Hb in transfusion therapy with LPS-challenged sepsis mouse model. Septic wild-type mice were transfused with RBCs from Presbyterian Hb-carrying mutant mice and wild-type mice. Their survival rates were assessed, and apoptosis of hepatocytes was evaluated. Survival rates of septic Presbyterian mutant mice and the wild-type littermates were also studied.

Results: The Presbyterian mutant RBC-transfused septic group survived longer than the wild-type RBC-transfused group. Apoptosis was reduced in the hepatocytes of the former group. Presbyterian mutant mice themselves, however, did not have stronger resistance to LPS-induced sepsis.

Conclusion: Transfusion of low-affinity Hb-containing RBCs has beneficial effects in septic mice.

MeSH terms

  • Animals
  • Apoptosis
  • Bacterial Infections / physiopathology
  • Bacterial Infections / therapy
  • Binding, Competitive
  • Disease Susceptibility
  • Endotoxemia / physiopathology
  • Endotoxemia / therapy*
  • Erythrocyte Transfusion*
  • Erythrocytes / metabolism
  • Hemoglobins, Abnormal / genetics*
  • Hepatocytes
  • Humans
  • Lipopolysaccharides
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic / blood*
  • Mice, Transgenic / genetics*
  • Mice, Transgenic / physiology
  • Oxygen / blood
  • Survival Analysis

Substances

  • Hemoglobins, Abnormal
  • Lipopolysaccharides
  • hemoglobin Presbyterian
  • Oxygen