Dual effect of pyruvate in isolated nerve terminals: generation of reactive oxygen species and protection of aconitase

Neurochem Res. 2005 Oct;30(10):1331-8. doi: 10.1007/s11064-005-8805-0.

Abstract

Generation of reactive oxygen species (ROS) in synaptosomes was investigated in the presence of different substrates. When pyruvate was used as a substrate an increased rate of hydrogen peroxide formation was detected by the Amplex Red fluorescent assay, but aconitase, which is known to be a highly sensitive enzyme to ROS was not inhibited. In contrast, pyruvate exerted a partial protection on aconitase against a time-dependent inactivation that occurred when synaptosomes were incubated in the absence of substrates. Disruption of synaptosomal membranes with Triton X-100 prevented the protective effect of pyruvate. It is suggested that citrate and/or isocitrate formed in the metabolism of pyruvate could be responsible for a partial protection of aconitase. Therefore while pyruvate could have a prooxidant effect it could also exert a protective effect on the aconitase.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aconitate Hydratase / metabolism*
  • Animals
  • Glucose / metabolism
  • Guinea Pigs
  • Hydrogen Peroxide / metabolism
  • Ketoglutaric Acids / metabolism
  • Neurons / cytology
  • Neurons / metabolism*
  • Neuroprotective Agents / metabolism*
  • Oxidants / metabolism
  • Pyruvic Acid / metabolism*
  • Reactive Oxygen Species / metabolism
  • Synaptosomes* / chemistry
  • Synaptosomes* / metabolism

Substances

  • Ketoglutaric Acids
  • Neuroprotective Agents
  • Oxidants
  • Reactive Oxygen Species
  • Pyruvic Acid
  • Hydrogen Peroxide
  • Aconitate Hydratase
  • Glucose