Regulation of graft-versus-host-reaction by Mlsa-reactive donor T cells

Eur J Immunol. 1992 Aug;22(8):1967-73. doi: 10.1002/eji.1830220803.

Abstract

Injection of A/J splenocytes (H-2Dd, Mlsc) into unirradiated (A/J x CBA)F1 (BAF1) host mice (H-2Dd/k, Mlsd) results in an acute suppressive graft-vs.-host reaction (GVHR), characterized by immune dysfunction and appreciable donor cell engraftment; injection of the CBA/J parent (H-2Dk, Mlsd), which recognizes no Mls disparity in the host, results in little or no GVHR. Furthermore, the Mlsa-reactive V beta 6 and V beta 8.1 T cell subsets in A/J T cells expand significantly in the GVHR host. Finally, depletion of V beta 6+ and V beta 8.1+ from the A/J population abrogates the proliferative response to BAF1 in vitro and the development of GVHR in vivo. Thus, the response to Mls determinants can contribute to the generation of a GVHR.

MeSH terms

  • Animals
  • Graft vs Host Reaction*
  • Lymphocyte Depletion
  • Mice
  • Mice, Inbred C57BL
  • Mice, Inbred CBA
  • Minor Lymphocyte Stimulatory Antigens / physiology*
  • T-Lymphocyte Subsets / physiology
  • T-Lymphocytes / physiology*

Substances

  • Minor Lymphocyte Stimulatory Antigens