The mitochondria and insulin release: Nnt just a passing relationship

Cell Metab. 2006 Jan;3(1):5-7. doi: 10.1016/j.cmet.2005.12.003.

Abstract

Nicotinamide nucleotide transhydrogenase (Nnt) detoxifies reactive oxygen species (ROS), byproducts of mitochondrial metabolism that, when accumulated, can decrease mitochondrial ATP production. In this issue of Cell Metabolism, demonstrate that Nnt in pancreatic beta cells is important for insulin release. Their compelling data highlight the critical roles for ATP generation and subsequent closure of KATP channels for insulin secretion.

Publication types

  • Comment

MeSH terms

  • Animals
  • Insulin / metabolism*
  • Insulin Secretion
  • Insulin-Secreting Cells / enzymology
  • Insulin-Secreting Cells / metabolism
  • Mice
  • Mitochondria / enzymology*
  • Mitochondrial Proteins / genetics
  • Mitochondrial Proteins / physiology*
  • NADP Transhydrogenases / genetics
  • NADP Transhydrogenases / physiology*

Substances

  • Insulin
  • Mitochondrial Proteins
  • NADP Transhydrogenases