Role of increase in intracellular calcium in PTH-induced homologous desensitization in UMR-106 cells

Biochem Biophys Res Commun. 1991 May 15;176(3):1033-6. doi: 10.1016/0006-291x(91)90386-l.

Abstract

Effects of increase in intracellular calcium on PTH-induced homologous desensitization were investigated using calcium ionophores. Pretreatment of UMR-106 cells (rat osteoblast like osteosarcoma cell line) with calcium ionophores (A23187 or ionomycin) for 6h resulted in approximately 50% decrease of PTH-stimulated cAMP production. PTH receptor binding, assessed with 125I-[Nle8,Nle18,Tyr34]PTH-(1-34) as radioligand, was significantly decreased in 10(-6) M calcium ionophore-pretreated (for 6h) cells without affecting the dissociation constant (Kd) for PTH. Minimal effective treatment period was 2h and similar inhibitory effect was observed in 12h-treated cells. These data suggest that increase in intracellular calcium might also act on PTH receptor in the similar manner as protein kinase C activation to induce desensitization.

MeSH terms

  • Animals
  • Calcimycin / pharmacology
  • Calcium / metabolism*
  • Cell Line
  • Cyclic AMP / metabolism
  • Ionomycin / pharmacology
  • Kinetics
  • Osteoblasts / metabolism
  • Osteosarcoma
  • Parathyroid Hormone / metabolism
  • Parathyroid Hormone / pharmacology*
  • Rats
  • Receptors, Cell Surface / drug effects
  • Receptors, Cell Surface / metabolism*
  • Receptors, Parathyroid Hormone

Substances

  • Parathyroid Hormone
  • Receptors, Cell Surface
  • Receptors, Parathyroid Hormone
  • Calcimycin
  • Ionomycin
  • Cyclic AMP
  • Calcium