A novel nonsense mutation in the LPL gene in a Chinese neonate with hypertriglyceridemia

Clin Chim Acta. 2006 Jun;368(1-2):120-4. doi: 10.1016/j.cca.2005.12.020. Epub 2006 Feb 7.

Abstract

Background: Lipoprotein lipase (LPL) deficiency is a rare autosomal recessive disorder characterized by hypertriglyceridemia. The genetic defect lies in a mutation of the LPL gene.

Methods: A Chinese neonate with non-consanguineous parents was incidentally found to have hypertriglyceridemia. Mutation in her LPL gene was screened by using polymerase chain reaction and direct DNA sequencing.

Results: Homozygous missense mutations (L252V) were detected in the LPL gene of the patient. A novel nonsense mutation (C27X) was also identified.

Conclusion: Our finding supports L252V mutation in the LPL gene is a common mutation in Chinese with familial hyperchylomicronemia syndrome. DNA-based diagnosis in this syndrome is definitive. It saves the need for heparin-infusion test, which carries the risk of hemorrhage, and the measurement of LPL activity, which is tedious and is not widely available.

Publication types

  • Case Reports

MeSH terms

  • Asian People
  • Base Sequence
  • Codon, Nonsense / genetics*
  • Cysteine / genetics
  • Female
  • Genotype
  • Humans
  • Hypertriglyceridemia / congenital*
  • Hypertriglyceridemia / genetics*
  • Infant, Newborn
  • Leucine / genetics
  • Lipid Metabolism
  • Lipoprotein Lipase / genetics*
  • Male
  • Pedigree

Substances

  • Codon, Nonsense
  • Lipoprotein Lipase
  • Leucine
  • Cysteine