EGFR, c-erbB-2 and ki-67 in NSCLC and preneoplastic bronchial lesions

Anticancer Res. 2006 Jan-Feb;26(1A):135-8.

Abstract

Background: The relationships between EGF-R and c-erbB-2 with other factors involved in tumour regulation are not well understood. The aim of this study was to correlate the expression of these markers with tumour proliferation.

Materials and methods: The presence of EGF-R, c-erbB-2 and Ki-67 was evaluated by immunohistochemistry in non-small cell lung cancer (NSCLC) and preneoplastic lesions.

Results: Forty-two percent of the tumours were positive for EGF-R, 22% for c-erbB-2 and 97% for Ki-67. No statistically significant correlation was found between EGF-R and Ki-67, EGF-R and c-erbB-2 or between c-erbB-2 and Ki-67. With regards to Ki-67, a significant difference in survival was noted in favour of patients who did not express the marker. In preneoplastic lesions, most of the low-grade lesions showed neither EGF-R nor Ki-67 staining. In contrast, most of the high-grade lesions stained positively for these proteins.

Conclusion: EGF-R and c-erbB-2 do not seem to be correlated with Ki-67 in NSCLC.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Biomarkers, Tumor / biosynthesis
  • Biopsy
  • Bronchial Neoplasms / metabolism*
  • Bronchial Neoplasms / pathology
  • Bronchial Neoplasms / surgery
  • Carcinoma, Non-Small-Cell Lung / metabolism*
  • Carcinoma, Non-Small-Cell Lung / pathology
  • Carcinoma, Non-Small-Cell Lung / surgery
  • Cell Growth Processes / physiology
  • ErbB Receptors / biosynthesis*
  • Female
  • Humans
  • Immunohistochemistry
  • Ki-67 Antigen / biosynthesis*
  • Lung Neoplasms / metabolism*
  • Lung Neoplasms / pathology
  • Lung Neoplasms / surgery
  • Male
  • Middle Aged
  • Receptor, ErbB-2 / biosynthesis*

Substances

  • Biomarkers, Tumor
  • Ki-67 Antigen
  • ErbB Receptors
  • Receptor, ErbB-2