Advantage of ischemic preconditioning for hepatic resection in pigs

J Surg Res. 2006 Aug;134(2):173-81. doi: 10.1016/j.jss.2006.02.001. Epub 2006 Mar 20.

Abstract

Background: Ischemic preconditioning (IP) and intermittent inflow occlusion (IO) have provided beneficial outcomes in hepatic resection. However, comparison of these two procedures against warm hepatic ischemia-reperfusion injury has not been studied enough.

Materials and methods: Pigs that had undergone 65% hepatectomy were subjected to Control (120 min continuous ischemia, n = 6), IP (10 min ischemia and 10 min reperfusion, followed by 120 min continuous ischemia, n = 6), and IO (120 min ischemia in the form of eight successive periods of 15 min ischemia and 5 min reperfusion, n = 6). We evaluated hepatocyte injury by aspartate aminotransferase, lactate dehydrogenase and hepaplastin test, hepatic microcirculation by hepatic tissue blood flow (HTBF) and endothelin (ET)-1, inflammatory response by tumor necrosis factor-alpha (TNF-alpha), and histopathology after reperfusion.

Results: IP prevented hepatocyte injury, HTBF disturbance, and hepatocyte necrosis in histopathology as well as IO. These two groups showed significantly better outcomes than Control. IP produced significantly less ET-1 and TNF-alpha than IO.

Conclusions: IP ameliorated hepatic warm ischemia-reperfusion injury. Furthermore, IP gained more advantages in preventing chemokine production such as ET-1 and inflammatory response over IO. IP could take the place of IO for hepatectomy.

MeSH terms

  • Animals
  • Aspartate Aminotransferases / blood
  • Blood Flow Velocity
  • Endothelin-1 / blood
  • Hepatectomy / methods
  • Inflammation / pathology
  • Ischemic Preconditioning*
  • L-Lactate Dehydrogenase / blood
  • Liver / blood supply
  • Liver / pathology
  • Liver / surgery*
  • Male
  • Microcirculation
  • Necrosis
  • Postoperative Complications
  • Reperfusion Injury / pathology
  • Reperfusion Injury / prevention & control
  • Specific Pathogen-Free Organisms
  • Swine
  • Time Factors
  • Tumor Necrosis Factor-alpha / analysis

Substances

  • Endothelin-1
  • Tumor Necrosis Factor-alpha
  • L-Lactate Dehydrogenase
  • Aspartate Aminotransferases