MAP kinase pathway-dependent phosphorylation of the L1-CAM ankyrin binding site regulates neuronal growth

Mol Biol Cell. 2006 Jun;17(6):2696-706. doi: 10.1091/mbc.e06-01-0090. Epub 2006 Apr 5.

Abstract

The growth of neuronal processes depends critically on the function of adhesion proteins that link extracellular ligands to the cytoskeleton. The neuronal adhesion protein L1-CAM serves as a receptor for nerve growth-promoting proteins, a process that is inhibited by the interaction between L1-CAM and the cytoskeleton adaptor ankyrin. Using a novel reporter based on intramolecular bioluminescence resonance energy transfer, we have determined that the MAP kinase pathway regulates the phosphorylation of the FIGQY motif in the adhesion protein L1-CAM and its interaction with ankyrin B. MAP kinase pathway inhibitors block L1-CAM-mediated neuronal growth. However, this blockade is partially rescued by inhibitors of L1-CAM-ankyrin binding. These results demonstrate that the MAP kinase pathway regulates L1-CAM-mediated nerve growth by modulating ankyrin binding, suggesting that nerve growth can be regulated at the level of individual receptors.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Ankyrins / antagonists & inhibitors
  • Ankyrins / metabolism*
  • Binding Sites
  • Cell Line, Tumor
  • Humans
  • MAP Kinase Signaling System / physiology*
  • Neural Cell Adhesion Molecule L1 / metabolism*
  • Neurites / physiology*
  • Neuroblastoma
  • Phosphorylation

Substances

  • Ankyrins
  • Neural Cell Adhesion Molecule L1