Avarol restores the altered prostaglandin and leukotriene metabolism in monocytes infected with human immunodeficiency virus type 1

Virus Res. 1991 Nov;21(3):213-23. doi: 10.1016/0168-1702(91)90034-s.

Abstract

Infection of monocytes with human immunodeficiency virus type 1 (HIV-1) (strain Ada-M) caused increased levels of leukotriene B4 (LTB4) and prostaglandin E2 (PGE2) in vitro. These two products result from the activities of the two enzymes cyclooxygenase and 5-lipoxygenase. The addition of the sesquiterpenoid hydroquinone Avarol, an HIV inhibitor, strongly reduced the levels of LTB4 and PGE2 via inhibition of both cyclooxygenase and lipoxygenase in monocytes. The 50% inhibition concentrations (IC50) for the enzymes were determined to be 2.26 microM (cyclooxygenase) and 1.97 microM (lipoxygenase). A 50% reduction of the extent of PGE2 and LTB4 production in HIV-infected monocytes was measured at a concentration of 0.9 microM Avarol, a dose which caused an 80% anti-HIV effect in vitro (50% inhibition of virus release from infected cells: 0.3 microM). We conclude that Avarol inhibits the enzymes cyclooxygenase and lipoxygenase and suggest that, in general, inhibitors of these enzymes are promising anti-HIV compounds.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antiviral Agents / pharmacology*
  • Arachidonate 5-Lipoxygenase / metabolism
  • Cyclooxygenase Inhibitors / pharmacology
  • Dinoprostone / metabolism*
  • HIV-1 / drug effects
  • HIV-1 / physiology*
  • Leukotriene B4 / metabolism*
  • Lipoxygenase Inhibitors / pharmacology
  • Monocytes / metabolism
  • Monocytes / microbiology*
  • Prostaglandin-Endoperoxide Synthases / metabolism
  • Sesquiterpenes / pharmacology*

Substances

  • Antiviral Agents
  • Cyclooxygenase Inhibitors
  • Lipoxygenase Inhibitors
  • Sesquiterpenes
  • Leukotriene B4
  • Arachidonate 5-Lipoxygenase
  • Prostaglandin-Endoperoxide Synthases
  • Dinoprostone
  • avarol